New Link Between Diet and Cancer
Diet. Inflammation. Cancer.
There's a link.
A connection between inflammation and cancer has been found, and diet and nutrition contribute to not only colitis but colitis-associated colon cancer. Chronic inflammation appears to play a key role in the development of cancer, along with heart disease and diabetes. Now a new study presented today suggests that eating a diet high in sugar, saturated fats and others foods that promote inflammation increases the risk of premature death from any cause, including cancers of the gastrointestinal tract.
A connection between inflammation and cancer has been recognized for over a hundred years, according to newswise.com. This connection is particularly evident in colon carcinogenesis, because patients with inflammatory bowel disease (IBD) have a higher incidence of colon cancer than the general population.
The Web site reports that a new study has found that there's increasing evidence that inflammation contributes to the earliest stages of carcinogenesis, namely in the process of cell transformation, where the cell acquires many aspects of cancer characteristics. The observation that IBD and colon cancer incidence rise as nations industrialize suggests that changes in diet and nutrition contribute to colitis and colitis-associated colon cancer, newswise states.
The study discovered that a category of lipids, the building blocks that make up the function and structure of living cells, play fundamental roles in carcinogenesis through their ability to regulate programmed cell death pathways, stress responses, immunity, and inflammation.
The final breakdown product of these lipids is a pro-inflammatory signaling lipid that promotes cell growth and carcinogenesis.
The research suggests that, while these special mammalian lipids may promote inflammation and carcinogenesis, plant/soy lipids (soybeans, for example) reduce the activity of several cancer signaling pathways, suggesting that dietary lipids may enhance or inhibit colon carcinogenesis, depending on their ability to be metabolized to an enzyme that protects against the formation of cancer cells.
All this to say that now, more than ever, diet matters, when it comes to cancer.
There's a link.
A connection between inflammation and cancer has been found, and diet and nutrition contribute to not only colitis but colitis-associated colon cancer. Chronic inflammation appears to play a key role in the development of cancer, along with heart disease and diabetes. Now a new study presented today suggests that eating a diet high in sugar, saturated fats and others foods that promote inflammation increases the risk of premature death from any cause, including cancers of the gastrointestinal tract.
A connection between inflammation and cancer has been recognized for over a hundred years, according to newswise.com. This connection is particularly evident in colon carcinogenesis, because patients with inflammatory bowel disease (IBD) have a higher incidence of colon cancer than the general population.
The Web site reports that a new study has found that there's increasing evidence that inflammation contributes to the earliest stages of carcinogenesis, namely in the process of cell transformation, where the cell acquires many aspects of cancer characteristics. The observation that IBD and colon cancer incidence rise as nations industrialize suggests that changes in diet and nutrition contribute to colitis and colitis-associated colon cancer, newswise states.
The study discovered that a category of lipids, the building blocks that make up the function and structure of living cells, play fundamental roles in carcinogenesis through their ability to regulate programmed cell death pathways, stress responses, immunity, and inflammation.
The final breakdown product of these lipids is a pro-inflammatory signaling lipid that promotes cell growth and carcinogenesis.
The research suggests that, while these special mammalian lipids may promote inflammation and carcinogenesis, plant/soy lipids (soybeans, for example) reduce the activity of several cancer signaling pathways, suggesting that dietary lipids may enhance or inhibit colon carcinogenesis, depending on their ability to be metabolized to an enzyme that protects against the formation of cancer cells.
All this to say that now, more than ever, diet matters, when it comes to cancer.
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